TELE MEDICINE

This is a de-identified patient online record with information interpreted from audio call with the patient, posting here after attaining verbal consent from the patient,by student doctor(Saahithya-141, Nikitha- 130, Sai Bhavana - 146) under the guidence of Dr Rakesh Biswas sir.

A 45 year male patient who works as a political leader has come to hospital with 
Chief complaints:
 Right sided chest pain since may 3rd.

History Of Present Illness
Patient was apparently asymptomatic before may 3rd. He developed localized pain which is on  right side of mid-line. It was insidious in onset, which is dull aching type and non radiating. Pain was gradually progressive for  3 days and remained persisted. Pain is aggravated on mild movement,coughing and inspiration. Pain was relieved on medication.He used to sleep only on one side due to pain and was not able to move even a little bit during sleep.It was associated with some episodes of pricking type of pain on back side just opposite to the localized pain in the ant chest wall.Pricking types pain was insidious in onset and lasted for small duration (2-3 secs for every 4 hours).He felt mild stony hard swelling on palpation at the site of localized pain.
He also has a history of pain on the shoulder,arm, little finger and ring finger( tingling was present).

He told that during the lock-down period as he did not have any work, he used to play carroms in the mobile for 12 -15 hours for 2 months continuously. He plays the game in the supine position with 2 to 3 pillows under the neck with left arm  resting on the bed and stains his  right upper limb while playing.COULD IT BE THE REASON BEHIND HIS CHEST PAIN?????

He also told that he had a history of mild neck pain since 10-15 years which may be due to use of 2-3 pillows.
 
There was a history of accident 15 years back (bike hit on the lower back). Pain was present in the lower back region and was relived after using medications. But some episodes of back pain are still present.He also had tingling sensation in lower limbs while squatting for 10 min(Is it normal??)

He also had history of hawking (frequent clearing of throat). He also gave a history of burning sensation in the central of chest and upper central abdomen after taking oily and fatty meals (which is relived by taking ENO) since 16 yrs. He told that he never felt hungry and he takes only two meals a day. For this he visited hospital and  was diagnosed with acidity. He didn't use medications, as he couldn't take meal properly in normal timing. After lock-down he started taking meals 3 times a day and stated walking for 3-4 kms everyday. Know he says there is no acidity.

No history of recent trauma
No history of palpitations,giddiness,SOB,headache,burning sensation
No history of fullness and squeezing.
No history of edema
No history of cough,cold, hemoptysis, wheeze
No history of anxiety
No history of redness, skin changes 

Treatment history:
Then patient developed mild pain on the right side of the right side for which he visited local rpm,  where he was treated for 15 days in clinic. Then he visited a cardiologist after 15 days onset of pain and it was relieved by medication within 4 days.He still has few episodes of pain which only relieved after taking medications.

Medical history
There is no history of similar complaints in past. Not a known case of hypertension, coronary artery diseases, diabetes mellitus, thyroid problems.
He has history of acid indigestion since 16 yrs.

Drug history
He has been using ENO, when he gets burning sensation after eating.
Tab Met XL 25mg
Tab Mactor asp 
Tab Pantotas
Tab Aceclo plus
Tab Paracetamol 650mg
Following medication have been prescribed by rmp 
Tab Ultracet
Tab Hifenac -P 
Tab Nexpro-40
Tab Hexagab SR
Tab Rabegard
Tab Beplex
Tab Montair

Family history
No significant family history

Personal history
Diet: mixed type and consumes high fatty diet(suspect of hyperlipidemia)
Appetite:decreased appetite and significant increase in appetite during lock-down
Sleep:adequate
Bowel and bladder movement: regular
Addictions:chronic alcoholic (weekly 3 times and each time 2 beer). He stopped drinking completely since 8 months.


SITE OF PAIN


INVESTIGATION
















ECG: V3 - biphasic T wave
Lead 1 ,2V1, V2, V4, V5, V6 show T wave inversion
Left ventricular hypertrophy





Our thoughts in this case
  • Burning sensation in his chest and upper epigastrium seems like GERD but could as be silent MI.
  • Pain in his right upper limb and right chest region may be excessive use of smart phone in abnormal position.
  • Lower back pain and tingling sensation in the squatting position may be due to accident causing compression of nerves.
  • It could be costochondritis as there is tenderness on palpation.
  • We can also think of Tietze syndrome as patient has swelling at the area of tenderness
  • In investigation TMT was positive(292bpm).This may be due to compensatory increase in heart rate due some underlying cardiac pathology.A study shown that,in some patients, a false positive TMT can be seen in obstructive coronary artery disease
https://academic.oup.com/eurheartj/article/34/suppl_1/P3375/2861932
  • As our patient consumes high fatty diet there may be atherosclerosis in the vessels.No other risk factors like smoking, hypertension,DM are present.Other than lack of physical activity,age, alcohol
  • According to the ECG and 2D ECHO we think of left ventricular hypertrophy.
  • As there are inverted T waves in V1,V2,V4,V5,V6 ,we think that there maybe myocardial ischemia.
INVESTIGATION REQUIRED ARE
  • Coronary angiogram
  • lipid profile

DIFFERENTIAL DIAGNOSIS

  • Left ventricular hypertrophy
  • Silent MI
  • Costochondritis/Tietze
  • GERD
QUERIES
  • Was osteophyte growth due to neck stain???
  • Is the tingling sensations in 4th and 5th digits of right upper limb is due to osteophyte growth causing C8nerve ( C8 RADICULOPATHY).
  • Is the left ventricular hypertrophy due to hypertrophic cardiomyopathy??
  • Did ALCOHOL effect the heart??

We thank our patient to be so cooperative and comfortable to talk to us on phone.

 My colleagues blog. Please take a look at them too .


Comments

  1. Student 1:
    5/27/20, 10:00 AM – UG 2016: Monomorphic P wave preceding each QRS complex tells that that there is sinus rhythm. Normal PR Interval. Normal QRS complex. Though, there appears to be tachycardia, but I don't know how to count/decipher without the boxes. Regular R-R interval. Axis is normal - (0 - 60 degrees), With a normal R wave progression in V1-V6
    5/27/20, 10:01 AM – UG 2016: Though there appears to be a wave before P wave in lead ll
    5/27/20, 10:01 AM – UG 2016: Sir what else do i look for ?
    5/27/20, 10:03 AM – UG 2106: Sir V4, V5, V6 donot appear normal
    5/27/20, 10:03 AM – GM Dept: Which ECG are you describing?
    5/27/20, 10:04 AM – UG 2016: Sir the one you sent on the group
    5/27/20, 10:05 AM – GM Dept: Good. Yes what is wrong here.
    5/27/20, 10:06 AM – UG 2016: ST depression ?
    5/27/20, 10:08 AM – GM Dept : And T wave?
    5/27/20, 10:08 AM – UG 2016: Inversion

    5/27/20, 10:10 AM – UG 2016 : T wave inversion is symmetrical or unsymmetrical sir ? Also sir, will the amplitudes of R wave in V5 and V6 added up to more than 35mm ? If they do, it could be LVH.
    5/27/20, 10:12 AM – GM Dept: Yes he has coronary artery disease and yet it's stable angina or even silent angina.
    Why? Because the ECG is not a resting ECG but taken after six minutes of exercise signifying a coronary artery blockage but he was never symptomatic due to it.
    His current 20 days of chest pain is on the right side and I just pressed on his right chest where he pointed to the pain and found it to be very tender suggesting a musculoskeletal chest pain

    5/27/20, 10:18 AM – UG 2016: Yes sir, stress ECG is a must as, at rest, the heart's compensatory mechanisms would be in action. Sir, is there a past history to any chronic disorders, in this patient ? And any finding in the blood tests ?
    5/27/20, 10:20 PM – GM Dept: Ecg taken today
    5/28/20, 8:33 AM – UG 206: Good morning Sir, this ECG and 2D ECHO are of the pt with chest tenderness on rt side ?
    5/28/20, 9:01 AM – GM Dept: Yes
    5/28/20, 9:13 AM – UG 2016: Sir there is ST depression, T inversion and left Ventricular hypertrophy (as the sum of amplitude of R wave in V5, V6 > 35mm ). Other than that, i cannot seem to see anything wrong, HR = 70 b/m
    5/28/20, 9:14 AM – UG 2016: And ST depression doesn't seem that significant. But since we saw it in the stress ECG, it becomes clearer.
    5/28/20, 9:14 AM – UG 2016: And sir, I am not being able to understand the 2D echo.
    5/28/20, 9:19 AM – GM Dept: The main feature in that echo is the gross LVH which alone accounts for the ECG findings. In the absence of any significant hypertension this would be labeled as hypertrophic cardiomyopathy.

    5/28/20, 9:24 AM – UG 2016: Thank you sir. Does he have a h/o any chronic medical condition or medications which might have caused Hypertrophic Cardiomyopathy ?
    5/28/20, 10:20 AM – GM Dept : No. Didn't get a chance to document that.



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  2. Student 2:
    "[5/27, 10:57 AM] MBBS 2016 UG 5: Good morning sir!
    [5/27, 10:57 AM] MBBS 2016 UG 5: Maybe the pain and the ECG are unrelated because he has the pain since 20 days
    [5/27, 10:58 AM] MBBS 2016 UG 5: And usually pains related to the heart don't last that long or goes away when they take medications or deep breaths
    [5/27, 10:17 PM] Post residency PG1:Yes he has coronary artery disease and yet it's stable angina or even silent angina.
    Why?
    Because the ECG is not a resting ECG but taken after six minutes of exercise signifying a coronary artery blockage but he was never symptomatic due to it.
    His current 20 days of chest pain is on the right side and I just pressed on his right chest where he pointed to the pain and found it to be very tender suggesting a musculoskeletal chest pain
    [5/28, 9:13 AM] MBBS 2016 UG 5: Sir so due to the ischemia he might've developed a muscle pain?
    [5/28, 9:14 AM] MBBS 2016 UG 5:Also sir. The fire accident is very disturbing. I hope no one was injured.
    [5/28, 9:18 AM] Post residency PG1: No ischemia to myocardium can't cause chest muscle pain.
    However if you check out the Echo then our initial assumptions of ischemia may not be true in accounting for his Ecg findings. The anatomic localization for those Ecg findings may shift from coronary disease to elsewhere in the heart
    [5/28, 9:23 AM] MBBS 2016 UG :Okay sir.
    Sir any chance we can maybe suspect any costochondritis?

    [5/28, 9:24 AM] MBBS 2016 UG 5: Did he have any history or trauma or muscle strain?
    [5/28, 10:01 AM] Post residency PG1:Yes that was the number one diagnosis for his pain due to the extreme tenderness noted in the right side of his chest.
    So this case illustrates that the patient can have Ecg findings due to a completely different reason whereas the chest pain symptoms could be due to a different reason"
    28/05/20, 10:06:27 AM] MBBS 2016 UG 5: Okay sir.
    Sir, I wanted to know, why was his initial ECG without the normal squares? Like why is that done?
    [28/05/20, 10:06:51 AM] MBBS 2016 UG 5: Okay sir.
    [28/05/20, 10:07:45 AM] Post residency PG1: That was a print out where they managed to blur the squares
    [28/05/20, 10:10:36 AM] MBBS 2016 UG 5 : 28/05/20, 10:11:09 AM] :Why is that done sir?
    Isn't it better to interpret with the squares?
    [28/05/20, 10:15:12 AM] Post residency PG1:ECGs are done on heat sensitive paper and are more of heat lines that can quickly vanish with time. Print outs as photocopies in regular paper are taken to increase the longevity of the ECG information and these print outs may miss the squares?
    [5/28, 11:30 AM] MBBS 2016 UG 5: Sir. Has the patient been discharged or is he undergoing any confirmatory tests?
    [5/28, 11:31 AM] MBBS 2016 UG 5
    : Because to confirm Costchondritis, the CRp should be raised

    [5/28, 11:31 AM] Post residency PG1: He had just come for an OPD. Stays nearby
    [5/28, 11:32 AM] Post residency PG1: CRP is a non specific marker of inflammation
    [5/28, 11:33 AM] MBBS 2016 UG 5
    : Sir in this blog there are many patients who inspite of having HOCM have always been diagnosed with costochondritis
    [5/28, 11:34 AM] Post residency PG1: Excellent find. Share the blog link so that it's easier to put on the active learning blog
    [5/28, 11:34 AM] MBBS 2016 UG 5:
    https://messageboard.4hcm.org/forum/hcma-general-forums/hcma-discussion/2918-costochondritis
    [5/28, 11:35 AM] MBBS 2016 UG 5:
    : So was the patient given anti inflammatory medication sir?
    [5/28, 11:35 AM] Post residency PG1: Yes

    ReplyDelete

  3. Student 3:

    [5/27, 1:31 PM] MBBS 2016 UG1: Does he have a history of fever?
    [5/27, 1:33 PM] MBBS 2016 UG1: What is the character of his Chest pain? Does it vary with respiration? Is it relieved on sitting and leaning forward?
    [5/27, 10:19 PM] POST RESIDENCY PG1: Didn't ask
    [5/27, 10:20 PM] POST RESIDENCY PG 1: Ecg taken today : (image)



    [5/27, 10:27 PM] MBBS 2016 UG1: In V3, there appears to be an ST elevation and a U wave?
    [5/27, 10:29 PM] MBBS 2016 UG1: Yes. Have you looked at the Echo? Just your knowledge of anatomy and physiology should be enough to interpret it

    [5/27, 10:33 PM] MBBS 2016 UG1: Mitral regurgitation?

    [5/27, 10:36 PM] POST RESIDENCY PG1: No prominent left ventricular hypertrophy and that is the reason for his ECG changes even more than possibility of coronary artery disease. Even mild ST elevation that you pointed out is indicative of LVH
    [5/27, 10:36 PM] MBBS 2016 UG1 What is the reason sir?
    [5/27, 10:45 PM] POST RESIDENCY PG1: Didn't check his BP. If not hypertension then HOCM
    [5/27, 10:45 PM] MBBS 2016 UG1: Oh okay. Thank you.
    [5/28, 10:03 AM] MBBS 2016 UG1: Could this patient also have mitral regurg and/or SAM. Patients with HCM frequently have systolic anterior motion (SAM) of the mitral valve, which positions the mitral valve within the LVOT.
    [5/28, 10:05 AM] MBBS 2016 UG1: And in this ECG, there appear to be some Deeply inverted T waves (so-called "giant negative T waves") - seen in V4 - V6 in patients with the apical variant of HCM.
    [5/28, 10:10 AM] POST RESIDENCY PG1: Possible. I shall have to do the echo myself to confirm that.
    However even if the patient doesn't have all that it would still be HCM if not HOCM.
    Even without the LVOT obstruction, HCM can be equally problematic in terms of morbidity causing heart failure

    ReplyDelete

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